參考文獻 |
Amaya F., Shinosato G., Nagano M., Ueda M., Hashimoto S., Tanaka Y., Suzuki H., and Tanaka M., 2004. NGF and GDNF differentially regulate TRPV1 expression that contributes to development of inflammatory thermal hyperalgesia. Eur. J. Neurosci. 20, pp. 2303-2310 .
Andrew D. and Greenspan J. D., 1999. Mechanical and heat sensitization of cutaneous nociceptors after peripheral inflammation in the rat. J. Neurophysiol., pp. 2649-2656.
Black J. A., Liu S., Tanaka M., Cummins T R., and Waxman S G., 2004. Change in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain. Proc. Natl. Acad. Sci. USA, 108, pp. 237-247.
Breese N. M., George A. C., Pauers L. E., and Stucky C. L., 2005. Peripheral inflammation selectively increases TRPV1function in IB4-positive sensory neurons form adult mouse. Pain, 115, pp. 37-49.
Caterina M. J., Leffler A., Malmberg A. B., Martin W. J., Trafton J., Petersen-Zeitz K. R., Koltzenburg M., Basbaum A. I. and Julius D.,2000. Impaired Nociception and pain sensation in mice lacking the capsaicin receptor. Science 288, pp. 306-313.
Chen C. C., Zimmer A., Sun W. H., Hall J., Bronstein M. J., and Zimmer A., 2002. A role for ASIC3 in the modulation of high-intensity pain stimuli. Proc. Natl. Acad. Sci., 99 (13), pp. 8992-8997.
Choi J. W., Lee S. Y. and Choi Y., 1996. Identification of a putative G protein-coupled receptor induced during activation –induced apoptosis of T-cells. Cell. Immunol., 168, pp. 78-84.
Costigan M. and Woolf C. J., 2000. Pain: Molecular mechanisms. J. Pain., 1, pp. 35-44.
Dirajlal S., Pauers L. E., and Stucky C. L., 2003. Differential responses properties of IB4-positve and –negative ummyelinated senspry neurons to protons and capsaicin. J. Neurophysiol. 89, pp. 513-524.
Djouhri L., Koutsikou S., Fang C., McMullan S., an Lawson S. N., 2006. Spontaneous pain, both neuropathic and inflammatory, is related to frequency of spontaneous firing in intact C-fiber nociceptors. J. Neurosci., 26 (4), pp. 1281-1292.
Gitterman D.P., Wilson L. and Randall A.D., 2005. Functional properties and pharmacological inhibition of ASIC channels in the human SJ-RH30 skeletal muscle cell line. J. Physiol., 562.3, pp. 759-769.
Huang J.W., 2005. The expression of proton-sensing G-protein-couopled receptor, OGR1, in pain-related neurons. Thesis.
Hucho T.B., Dina O. A., and Levine J. D., 2005. Epac mediates a cAMP-to PKC signaling in inflammatory pain: an ioslection B4 (+) neurons-specific mechanism. J. Neurosci. 25, pp. 6119-6126.
Hunt S.P., and Mantyh P. W., 2001. The molecular dynamics of pain control. Nature reviews, 2, pp. 83-90.
Im D. S., Heise C. E., Nguyen T., O’Dowd B. F. and Lynch K. R., 2001. Identification of a molecular target of psychosine and its role in globoid cell formation. J. Cell. Biol., 153, pp. 429-434.
Julius D. and Basbaum A. I., 2001. Molecular mechanisms of nociception. Nature, 413, pp. 203-210.
Kandel E.R., Schwartz J.H. and Jessell, T.M., Principles of neural science. 4th edition. Chapter 24.
Kanazawa T., Nakamura S., Momoi M., Yamaji T., Takematsu H., Yano H., Sabe H., Yamamoto A., Kawasaki T. and Kozutsumi Y., 2000. Inhibition of cytokinesis by a lipid metabolite, psychosine. J. Cell. Biol. 149, pp. 943-950.
Kinhart O., Obreja O., and Kress M., 2003. The inflammatory mediators serotonin, prostaglandinf E2 and bradykinin evoke calcium influx in rat sensory neurons. Neurosci., 188, pp. 69-74.
Lugwig M., Vanek M., Guerini D., Gasser J. A., Jones C.E., Hofstetter H., Wolf R. M. and Seuwen K., 2003. Proton-sensing G-protein-coupled receptors. Nature, 425, pp. 93-98.
Ma Q.P. and Woolf C. J., 1997. The progressive tactile hyperalgesia induced by peripheral inflammation is nerve growth factor dependent. NeuroReport, 8, pp. 807-810.
Mamet J., Lazdunski M., and Voiiley N., 2003. How nerve growth factor drives physiological and inflammatory expressions of Acid-sensing ion channel 3 in sensory neurons. J. Biol. Chem. 278 (49), pp. 48907-48913.
Menendez L., Lastra A., Hidalgo A. and Baamonde A., 2004. The analgesic effect induced by capsaicin is enhanced in inflammatory states. Life sciences, 74, pp. 3235-3244.
Mohapatra D. P. and Nau C., 2005. Regulation of Calcium-dependent desensitization in the canilloid receptor TRPV1 by calcineurin and cAMP-dependent protein kinase. J. Biol. Chem. 280, pp. 13424-13432.
Mogil J.S., Breese N.M., Witty M.-F., Ritchie J., Rainville M.-L., Ase A., Abbadi N., Stucky C.L. and Seguela P., 2005. Transgenic expression of a dominant-negative ASIC3 subunit leads to increased sensitivity to mechanical and inflammatory stimuli. J. of neurosci., 25(43), pp. 9893-9901.
Murakam N., Yokomizo T., Okuno T., and Shimizu T., 2004. G2A is a proton- sensing G-protein-couopled receptor antagonized by lysophosphatidalcholine. J. Biol. Chem. 279 (41), pp. 42484-42491.
O’Donnell J.M. snd Zhang H.-T., 2004. Antidepressant effects of inhibitors of cAMP phosphodiesterase (PDE4). Trends Pharmacol. Sci., 25 (3), pp. 158-163.
Parada C. A., Reichling D. B., and Levine J. D., 2005. Ghronic hyperalgesia priming in the rat involves a novel interaction between cAMP and PKCε second messenger pathway. Pain 113, pp. 185-190.
Reeh P. W. and Steen K. H., 1996. Tissue acidosis in nociception and pain. Brain research 113, pp. 143-151.
Ringkamp M., Peng Y B., Wu G., Harkte T. V. Campbell J. N., and Meyer R. A., 2001. Capsaicin responses in heat-sensitive and heat-insensitive A-fiber nociceptors. J. Neurpsci. 21 (12), pp. 4460-4468.
Sakai H.S., Lingueglia E., Champigny G., Mattei M.-G. and Lazdunski M., 1999. Cloning and functional expression of a novel degenerin-like Na+ channels gene in mammals. J. Physiol., 519-2, pp. 323-333.
Sawynok J., Reid A., and Meisner J., 2006. Pain behaviors produced by capsaicin: influence of inflammatory mediators and nerve injury. J. Pain. 7 (2), pp. 134-141.
Steen K.H., Reeh P.W., Anton F. and Handwarker H.O., 1992. Protons selectively induce lasting excitation and sensitization to mechanical stimulation of nociceptors in rat skin, in vitro. J. Nurosci. 12 (1), pp. 86-95.
Steen K.H., Steen A.E., Kreysel H.-W. and Reeh P.W., 1996. Inflamatory mediators potentiate pain induced by experimental tissue acidosis. Pain 66, pp. 163-170.
Stucky C. L., and Lewin G. R., 1999. Isolection B4-poitive and –negative nociceptors are functionally distinct. J. Neurosci. 19 (15), pp. 6497-6505.
Sutherland S. P., Benson C. J., Adelmen J. P. and McCleskey E.W., 2001. Acid-sensing ion cahnnels 3 matches the acid-gated current in cardiac ischemia-sensing neurons. Proc. Natl. Acad. Sci. USA, 98 (2), pp. 711-716.
Thompson S. W. N., Dray A., McCaeson K. E., Krause J. E., and Urban L., 1995. Nerve growth factor induces mechanical allodynia associated with novel A fiber-evoked spinal reflex activity and enhanced neurokinin-1 receptor activation in the rat. Pain 62, pp. 219-231.
Tomura H., Wang J-Q., Komachi M., Damirin A., Mogi C., Tobo M., Kon J., Misawa N., Sato K. and Okajima F., 2005. Prostaglandin I2 production and cAMP accumulation in response to acidic extracellular pH through OGR1 in human aortic smooth muscle cells. J. Biol. Chem. 280, pp. 34458-34464.
Tomura H., Mogi C., Sato K. and Okajima F., 2005. Proton-sensing and lysolipid-sensitive G-protein-coupled receptors: A novel type of multi-funcional receptors. Cell. Signal. 17, pp. 1466-1476.
Ugawa S., Ueda T., Nishigaki M., Shibata Y. and Shimada S., 2002. Amiloride-blockable acid-sensing ion channels are leading acid sensors expressed in human nociceptors. J. Clin. Invest. 110, pp. 1185-1190.
Usachev Y. and Verkhratsky A., 1995. IBMX induced calcium release from intracellular stores in rat sensory neurons. Cell Calcium 17, pp.197-206.
Wang, Z., Fluckiger, A. C., Nisitani, S., Wahl, M. I., Le, L. Q., Hunter, C. A., Fernal, A. A., Le Beau, M. M., and Witte, O. N., 2004. TDAG8 is a proton-sensing and psychosine-sensing G-protein-coupled receptor. J. Biol. Chem. 279, pp. 45626-45633.
Woolf C.J. and Salter M.W., 2000. Neuronal plasticity: increasing the gain in pain. Science 288, pp. 1765-1768.
Woolf C.J. amd Scholz J., 2002. Can we conquer pain? Nature. 5, pp. 1062-1067.
Zhu K., Baudhuin L M., Hong G., Williams F. S., Cristine K. L., Kabarowski J. H. S., Witte O N., and Xu Y., 2001. Sphingosylphosphorylcholine and lysophsphatidylcholine are ligands for the G protein-coupled receptor GPR4. J. Biol. Chem. 276 (44), pp. 41325-41335. |