參考文獻 |
1. Aley, K.O. and Levine, J.D. (1999). Role of protein kinace A in the maintenance
73
of inflammatory pain. The Journal of Neuroscience. 19: 2181-2186.
2. Aley, K.O., Messing. R.O., Mochly-Rosen, D., Levine, J.D. (2000). Chronic
hypersensitivity for inflammatory nociceptor sensitization mediated by the epsilon
isozyme of protein kinase C. The Journal of Neuroscience. 20 : 4680-4685.
3. Amadesi, S., Cottrell, G.S., Divino, L., Chapman, K., Grady, E.F., Bautista, F.,
Karanjia, R., Barajas-Lopez, C., Vanner, S., Vergnolle, N., Bunnett, N.W. (2006).
Protease-activated receptor 2 sensitizes TRPV1 by protein kinase Cepsilon- and
A-dependent mechanisms in rats and mice. J. Physiol. 575: 555-571.
4. An, S., Goetzl, E.J., Lee, H. (1998). Signaling mechanisms and molecular
characteristics of G protein-coupled receptors for lysophosphatidic acid and
sphingosine 1-phosphate. J. Cell Biochem Suppl. 30-31: 147-157.
5. Anliker, B., Chun, J. (2004). Cell surface receptors in lysophospholipid signaling.
Semin Cell Devl Biol. 15: 457-465.
6. Anliker, B., Chun, J. (2004). Lysophospholipid G Protein-coupled Receptors. J.
Biol. Chem. 279: 20555-20558.
7. Basbaum, A.I., Bautista, D.M., Scherrer, G., Julius, D. (2009) Cellular and
molecular mechanisms of pain. Cell. 139: 267-284.
8. Bevan, S. and Yeast, J. (1992). Protons activate a cation conductance in a
sub-population of rat dorsal root ganglion neurons. J. Physiol. 433: 145-161.
74
9. Bhave, G., Gereau, R.W. 4th. (2004) Posttranslational mechanisms of peripheral
sensation. The Journal of Neurobiology. 61: 88-106.
10. Bitar, K.N. and Yamata, H. (1995). Modulation of smooth muscle contraction
by sphingosylphosphorylcholine. American Journal of Phisiology. 269: 370-377.
11. Bockaert, J., Perroy, J., Becamel, C., Marin, P., Fagni, L. (2010). GPCR
interacting proteins (GIPs) in the nervous system: Roles in physiology and
pathologies. Annu Rev Pharmacol Toxicol. 50: 89-109.
12. Boguslawski, G., Lyons, D., Harvey, K.A., Kovala, A.T. and English, D. (2000).
Sphingosylphosphorylcholine Induces Endothelial Cell Migration and Morphogenesis.
Biochemical and Biophysical Research Communications. 272: 603-609.
13. Booden, M.A., Siderovski, D.P., Der, C.J. (2002). Leukemia-associated Rho
guanine nucleotide exchange factor promotes G alpha q-coupled activation of RhoA.
Mol Cell Biol. 22: 4053-4061.
14. Casey, B.J., Jones, R.M. (2010). Neurobiology of the adolescent brain and
behavior: implications for substance use disorders. J Am Acad Child Adolesc
Psychiatry. 49: 1189-1220.
15. Caterina, M.J., Schumacher, M.A., Tominaga, M., Rosen, T.A., Levine, J.D.,
Julius, D. (1997) The capsaicin receptor: a heat-activated ion channel in the pain
pathway. Nature. 389: 816-824.
75
16. Cesare, P., Dekker, L.V., Sardini, A., Parker, P.J., McNaughton, P.A. (1999).
Specific involvement of PKC-epsilon in sensitization of the neuronal response to
painful heat. Neuron. 23: 617-624.
17. Chisolm, G.M. and Steinberg, D. (2000). The oxidative modification hypothesis
of atherogenesis: an overview. Free Radical Biology and Medicine. 28: 1815-1826.
18. Clair, T., Aoki, J., Koh, E., Bandle, R.W., Nam, S.W., Ptaszynska, M.M., Mills,
G.B., Schiffmann, E., Liotta, L.A., Stracke, M.L. (2003). Autotaxin Hydrolyzes
Sphingosylphosphorylcholine to Produce the Regulator of Migration,
Sphingosine-1-Phosphate. Cancer Res. 63: 5446-5453.
19. Colles, S.M. and Chisolm, G.M. (2000). Lysophosphatidylcholine-induced
cellular injury in cultured fibroblasts involves oxidative events. J. Lipid Res. 41:
1188–1198.
20. Corey, D.P. and Garcia-Anoveros, J. (1996). Mechanosensation and the
DEG/ENaC ion channels. Science. 273: 361-364.
21. Davis, J.B., Gray, J., Gunthorpe, M.J., Hatcher, J.P., Davey, P.T., Overend, P.,
Harries, M.H., Latcham, J., Clappham, C., Atkinson, K., Hughes, S.A., Rance, K.,
Grau, E., Harper, A.J., Pugh, P.L., Rogers, D.C., Bingham, S., Randall, A.,
Sheardown, S.A. (2000). Vanilloid receptor-1 is essential for inflammatory thermal
hyperalgesia. Nature. 405: 183-187.
76
22. Desai, N.N., Carlson, R.O., Mattie, M.E., Olivera, A., Buckley, N.E., Seki, T.,
Brooker, G., Spiegel, S. (1993). Signaling Pathways for
Sphingosylphosphorylcholine-mediated Mitogenesis in Swiss 3T3 Fibroblasts. The
Journal of Cell Biology. 121: 1385-1395.
23. Ding, W.G., Toyoda, F., Ueyama, H., Matsuura, H. (2011).
Lysophosphatidylcholine enhances I(Ks) currents in cardiac myocytes through
activation of G protein, PKC and Rho signaling pathways. Journal of Molecular and
Cellular Cardiology. 50: 58-65.
24. Dray, A., Urban, L., Dickenson, A. (1994) Pharmacology of chronic pain.
Trends Pharmacol Sci. 12: 190-197.
25. Dray, A. (1995). Imflammatory mediators of pain. British Journal of
Anaesthesia. 75: 125-131.
26. Eglen, R.M., Bosse, R., Reisine, T. (2007). Emerging concepts of guanine
nucleotide-binding protein-coupled receptor (GPCR) function and implications for
high throughput screening. Assay Drug Dev Technol. 5: 425-451.
27. Hall, S.M., Gregson, N.A. (1971). The in Vivo and Ultrastructural Effects of
Injection of Lysophosphatidyl Choline into Myelinated Peripheral Nerve Fibres of the
Adult Mouse. J. Cell Sci. 9: 769-789.
28. Ichioka, T., Kishimoto, Y. and Yeager, A.M. (1987). Simultaneous
77
determination of psychosine and cerebrosides. Analytical Biochemistry. 166: 178-182.
29. Im, D., Heise, C.E., Nguyen, T., O’Dowd, B.F. and Lynch, K.R. (2001).
Identification of a molecular target of psychosine and its role in globoid cell
formation. J. Cell Biology. 153: 429-434.
30. Im, D. (2005). Two ligands for a GPCR, proton vs lysolipid. Acta
Pharmacologica Sinica. 26: 1435-1441.
31. Inoue, M., Rashid, M.H., Fujita, R., Contos, J.J., Chun, J., Ueda, H. (2004).
Initiation of neuropathic pain requires lysophosphatidic acid receptor signaling. Nat
Med. 10: 712-718.
32. Inoue, M., Ma, L., Aoki, J., Chun, J. and Ueda, H. (2008). Autotaxin, a synthetic
enzyme of lysophosphatidic acid (LPA), mediates the induction of nerve-injured
neuropathic pain. Molecular Pain. 4: 1-6.
33. Inoue, M., Ma, L., Aoki, J., Chun, J., Ueda, H. (2008). Autotaxin, a synthetic
enzyme of lysophosphatidic acid (LPA), mediates the induction of nerve-injured
neuropathic pain. Mol Pain. doi: 10.1186/1744-8069-4-6.
34. Inoue, M., Xie, W., Matsushida, Y., Chun, J., Aoki, J., Ueda, H. (2008).
Lysophosphatidylcholine induces neuropathic pain through an action of autotaxin to
generate lysophosphatidic acid. Neuroscience. 152: 296-298.
35. Inoue, M., Ma, L., Aoki, J., Ueda, H. (2008). Simultaneous stimulation of spinal
78
NK1 and NMDA receptors produces LPC which undergoes ATX-mediated
conversion to LPA, an initiator of neuropathic pain. J. Neurochem. 107: 1556-1565.
36. Ishii, I., Fukushima, N., Ye, X.Q., Chun, J. (2004). Lysophospholipid receptors -
signaling and biology. Annu. Rev. Biochem. 73: 321-354.
37. Jones, N.G., Slater, R., Cadiou, H., McNaughton, P. and McMahon, S.B. (2004).
Acid-induced pain and its modulation in humans. J. Neurosci. 24: 10974-10979.
38. Julius, D. and Basbaum, A.I. (2001). Molecular mechanisms of nociception.
Nature. 413: 203-210.
39. Kabarowski, J.H., Zhu, K., Le, L.Q., Witte, O.N., Xu, Y. (2001).
Lysophosphatidylcholine as a ligand for the immunoregulatory receptor G2A. Science.
293: 702-705.
40. Khasar, S.G., Gold, M.S., Levine, J.D. (1998). A tetratoxin-resistant sodium
current mediates inflammatory pain in the rat. Neurosci Lett. 256: 17-20.
41. Kidd, B.L., Urban, L.A. (2001). Mechanisms of inflammatory pain. Br J
Anaesth. 87: 3-11.
42. Kress, M., Reeh, P.W., Vyklicky, L. (1997). An interaction of inflammatory
mediators and protons in small diameter dorsal root ganglion neurons of the rat.
Neurosci. Lett. 224: 37-40.
43. Mair, N., Benetti, C., Andratsch, M., Leitner, M.G., Constantin, C.E.,
79
Camprubi-Robles, M., Quarta, S., Biasio, W., Kuner, R., Gibbins, I.L., Kress, M.,
Haberberger, R.V. (2011). Genetic Evidence for Involvement of Neuronally
Expressed S1P1 Receptor in Nociceptor Sensitization and Inflammatory Pain. PLoS
One. 6: 1-12.
44. Meyer zu Heringdorf, D., Himmel, H.M., Jakobs, K.H. (2002).
Sphingosylphosphorylcholine - biological functions and mechanisms of action.
Biochimica et Biophysica Acta. 1582: 178-189.
45. Millan, M.J. (1999). The induction of pain: an integrative review. Neurobiology.
192: 444-462.
46. Millan, M.J. (2002). Descending control of pain. Progress in Neurobiology. 66:
355-474.
47. Moolenaar, W.H. (1995). Lysophosphatidic Acid, a Multifunctional
Phospholipid Messenger. J. Biol. Chem. 270: 12949-12952.
48. Moriyana, T., Higashi, T., Togashi, K., Iida, T., Segi, E., Sugimoto, Y.,
Tominaga, T., Narumiya, S., Tominaga, M. (2005) Sensitization of TRPV1 by EP1
and IP reveals peripheral nociceptive mechanism of prostaglandins. Mol. Pain. 1: 3.
49. Murugesan, G. and Fox, P.L. (1996). Role of lysophosphatidylcholine in the
inhibition of endothelial cell motility by oxidized low density lipoprotein. J Clin
Invest. 97: 2736-2744.
80
50. Nishizuka, Y. (1992). Intracellular Signaling by Hydrolysis of Phospholipids
and Activation of Protein Kinase C. Science. 258: 6076-14.
51. Nixon, G.F., Mathieson, F.A., Hunter, I. (2008). The multi-functional role of
sphingosylphosphorylcholine. Prog Lipid Res. 47: 62-75.
52. Olah, Z., Karai, L., Iadarol,a M.J. (2002). Protein kinase C(alpha) is required for
vanilloid receptor 1 activation. Evidence for multiple signaling pathways. J Biol
Chem. 277: 35752-35759.
53. Pages, C., Simon, M.F., Valet, P., Saulnier-Blache, J.S. (2001).
Lysophosphatidic acid synthesis and release. Prostaglandins Other Lipid Mediat. 64:
1-10.
54. Pan, H.L., Zhang, Y.Q., Zhao, Z.Q. (2010). Involvement of lysophosphatidic
acid in bone cancer pain by potentiation of TRPV1 via PKC(epsilon) pathway in
dorsal root ganglion neurons. Pan et al. Molecular Pain. 6: 85-95.
55. Parada, C.A., Yeh, J.J., Reichling, D.B., Levine, J.D. (2003). Transient
attenuation of protein kinase Cepsilon can terminate a chronic hyperalgesic state in
the rat. Neuroscience. 120: 219-226.
56. Price, M.P., McIlwrath, S.L., Xie, J.H., Cheng, C., Qiao, J., Tarr, D.E., Sluka,
K.A., Brennan, T.J., Lewin, G.R., Welsh, M.J. (2001). The DRASIC Cation Channel
Contributes to the Detection of Cutaneous Touch and Acid Stimuli in Mice. Neuron.
81
32: 1071-1083.
57. Probst, W.C., Snyder, L.A., Schuster, D.I., Brosius, J., Sealfon, S.C. (1992).
Sequence alignment of the G-protein coupled receptor superfamily. DNA Cell Biol. 11:
1-20.
58. Pyne, S., Pyne, N. (2000). Sphingosine 1-phosphate signalling via the
endothelial differentiation gene family of G-protein-coupled receptors. Pharmacol
Ther. 88: 115-131.
59. Reichling, D.B., Levine, J.D. (2009). Critical role of nociceptor plasticity in
chronic pain. Trends Neurosci. 32: 611-618.
60. Sachs, D., Villarreal, C.F., Cunha, F.Q., Parada, C.A., Ferreira, S.H. (2009). The
role of PKA and PKC epsilon pathways in prostaglandin E2-mediated
hypernociception. British Journal of Pharmacology. 156: 826-834.
61. Sautel, M., Milligan, G. (2000). Molecular manipulation of G-protein-coupled
receptors: a new avenue into drug discovery. Curr Med Chem. 7: 889-896.
62. Scholz, J. and Woolf, C.J. (2002). Can we conquer pain? Nature Neuroscience.
5: 1062-1067.
63. Sluka, K.A., Kalra, A., Moore, S.A. (2001). Unilateral intramuscular injections
of acidic saline produce a bilateral, long-lasting hyperalgesia. Muscle Nerve. 24:
37-46.
82
64. Staniland, A.A., McMahon, S.B. (2009). Mice lacking acid-sensing ion channels
(ASIC) 1 or 2, but not ASIC3, show increased pain behaviour in the formalin test. Eur
J Pain. 13: 554-563.
65. Steen, K.H., Reeh, P.W., Anton, F. and Handwereker, H.O. (1992). Protons
selectively induce lasting excitation and sensitization to mechanical stimulation of
nociceptor in rat skin, in vitro. J. Neuroscience. 12: 86-93.
66. Steen, K.H., Steen, A.E. and Reeh, P.W. (1995). A Dominant Role of Acid pH
in Inflammatory Excitation and Sensitization of Nociceptors in Rat Skin, in vitro. J.
Neuroscience. 15: 3982-3985.
67. Steen, K.H. and Reeh, P.W. (1996). Tissue acidosis in nociception and pain.
Brain Research. 113: 143-151.
68. Steen, K.H., Steen, A.E., Kreysel, H.W., Reeh, P.W. (1996). Inflammatory
mediators potentiate pain induced by experimental tissue acidosis. Pain. 66: 163-170.
69. Strader, C.D., Fong, T.M., Graziano, M.P., Tota, M.R. (1995). The family of
G-protein-coupled receptors. FASEB J. 9: 745-754.
70. Sun, R.Q., Tu, Y.J., Lawand, N.B., Yan, J.Y., Lin, Q., Willis, W.D. (2004)
Calcitonin Gene-Related Peptide Receptor Activation Produces PKA- and
PKC-Dependent Mechanical Hyperalgesia and Central Sensitization. J Neurophysiol.
92: 2859-2866.
83
71. Taiwo, Y.O. and Levine, J.D. (1992). Serotonin is a directly-acting hyperalgesic
agent in the rat. Neuroscience. 48: 485-490.
72. van Meeteren, L.A., Ruurs, P., Christodoulou, E., Goding, J.W., Takakusa, H.,
Kikuchi, K., Perrakis, A., Nagano, T., Moolenaar, W.H. (2005). Inhibition of
autotaxin by lysophosphatidic acid and sphingosine 1-phosphate. J. Biol Chem. 280:
21155-21161.
73. Villarreal, C., Sachs, D., Cunha, F., Parada, C., Ferreira, S.H. (2009) The role of
PKA and PKCepsilon pathways in prostaglandin E2-mediated hypernociception. Br. J.
Pharmacol. 156: 826-834.
74. Voilley, N., Weille, J.D., Mamet, J., Lazdunski, M. (2001). Nonsteroid
anti-inflammatory drugs inhibit both the activity and the inflammation-induced
expression of acid-sensing ion channels in nociceptors. The Journal of Neuroscience.
21: 8026-8033.
75. Wakita, H., Matsushita, K., Nishimura, K., Tokura, Y., Furukawa, F. and
Takigawa, M. (1998). Sphingosylphosphorylcholine Stimulates Proliferation and
Upregulates Cell Surface-Associated Plasminogen Activator Activity in Cultured
Human Keratinocytes. Journal of Investigative Dermatology. 110: 253-258.
76. Walker, K.M., Urban, L., Medhurst, S.J., Patel, S., Panesar, M., Fox, A.J.,
McIntyre, P. (2003). The VR1antagonist capsaizepine reverses mechanical
84
hyperalgesia in models of inflammatory and neuropathic pain. Journal of
Pharmacology and Experimental Therapeutics. 304: 56-62.
77. Wallace, V.C., Cottrell, D.F., Brophy, P.J., Fleetwood-Walker, S.M. (2003)
Focal Lysolecithin-Induced Demyelination of Peripheral Afferents Results in
Neuropathic Pain Behavior That Is Attenuated by Cannabinoids. The Journal of
Neuroscience. 23: 3221-3233.
78. Welch, S.P., Sim-Selley, L.J., Selley, D.E. (2012). Sphingosine-1-phosphate
receptors as emerging targets for treatment of pain. Biochem Pharmacol. 84:
1551-1562.
79. Wu, J., Spiegel, S. and Sturgill, T.W. (1995). Sphingosine 1-phosphate rapidly
activates the mitogen-activated protein kinase pathway by a G protein-dependent
mechanism. J. Biol. Chem. 270: 11484-11488.1.
80. Xie, W., Strong, J.A., Kays, J., Nicol, G.D., Zhang, J.M. (2012). Knockdown of
the sphingosine-1-phosphate receptor S1PR1 reduces pain behaviors induced by local
inflammation of the rat sensory ganglion. Neurosci Lett. 515: 61-65.
81. Xu, Y., Zhu, K., Hong, G., Wu, W., Baudhuin, L.M., Xiao, Y., Damron, D.S.
(2000). Sphingosylphosphorylcholine is a ligand for ovarian cancer
G-protein-coupled receptor. Nat. Cell. Biol. 2: 261-267.
82. Zhu, K., Baudhuin, L.M., Hong, G., Williams, F.S., Cristina, K.L. (2001). Sphingosylphosphorylcholine and lysophosphatidylcholine are ligands for the G
protein-coupled receptor GPR4. J. Biol. Chem. 276: 41325-41335. |