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    Please use this identifier to cite or link to this item: http://ir.lib.ncu.edu.tw/handle/987654321/33538


    Title: 17 beta-Estradiol stimulates resistin gene expression in 3T3-L1 adipocytes via the estrogen receptor, extracellularly regulated kinase, and CCAAT/enhancer binding protein-alpha pathways
    Authors: Chen,Yen-Hang;Lee,Meng-Jung;Chang,Hsin-Huei;Hung,Pei-Fang;Kao,Yung-Hsi
    Contributors: 生命科學研究所
    Keywords: INSULIN-RESISTANCE;PLASMA RESISTIN;ADIPOSE-TISSUE;ADIPOGENESIS;ADIPONECTIN;ACTIVATION;OBESITY;LEPTIN;MOUSE;CELLS
    Date: 2006
    Issue Date: 2010-07-07 11:38:00 (UTC+8)
    Publisher: 中央大學
    Abstract: Resistin is known as an adipocyte-specific secretory hormone that can cause insulin resistance and decrease adipocyte differentiation. It can be regulated by sexual hormones, but the mechanism of estrogen's actions is still not clear. Using 3T3-L1 adipocy
    Relation: ENDOCRINOLOGY
    Appears in Collections:[Graduate Institute of Life Science] journal & Dissertation

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